REefsefaerccht of nutrient deficiencies on in vitro Th1 and Th2 cytokine response of peripheral blood mononuclear cells to Plasmodium falciparum infection
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BioMed Central
Abstract
Background: An appropriate balance between pro-inflammatory and anti-inflammatory cytokines that mediate
innate and adaptive immune responses is required for effective protection against human malaria and to avoid
immunopathology. In malaria endemic countries, this immunological balance may be influenced by micronutrient
deficiencies.
Methods: Peripheral blood mononuclear cells from Tanzanian preschool children were stimulated in vitro with
Plasmodium falciparum-parasitized red blood cells to determine T-cell responses to malaria under different conditions
of nutrient deficiencies and malaria status.
Results: The data obtained indicate that zinc deficiency is associated with an increase in TNF response by 37%; 95% CI:
14% to 118% and IFN-γ response by 74%; 95% CI: 24% to 297%. Magnesium deficiency, on the other hand, was
associated with an increase in production of IL-13 by 80%; 95% CI: 31% to 371% and a reduction in IFN-γ production.
These results reflect a shift in cytokine profile to a more type I cytokine profile and cell-cell mediated responses in zinc
deficiency and a type II response in magnesium deficiency. The data also reveal a non-specific decrease in cytokine
production in children due to iron deficiency anaemia that is largely associated with malaria infection status.
Conclusions: The pathological sequels of malaria potentially depend more on the balance between type I and type II
cytokine responses than on absolute suppression of these cytokines and this balance may be influenced by a
combination of micronutrient deficiencies and malaria status.
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Citation
Savelkoul, F. J. Effect of nutrient deficiencies on in vitro Th1 and Th2 cytokine response of peripheral blood mononuclear cells to Plasmodium falciparum infection. Nutritional zinc deficiency, immune capacity and malaria, 89.
